Why you shouldn't eat soy.

  • Thread starter Thread starter Ms.Wetback
  • Start date Start date
M

Ms.Wetback

Guest
Contrary to popular belief that soy is a health food, evidence reveals that soy consumption has been linked to numerous disorders, including infertility, increased cancer and infantile leukemia, Type1 diabetes, and precocious puberty in children have been fed soy formula. (early maturation, such as breast development and menstruation as early as 6 years of age). Scientists have known for years that the isoflavones in soy products can depress thyroid function and cause goiters in otherwise healthy children and adults. A combined research team of Cornell University Medical College and Long Island Community Hospital medical experts have found that children who develop Type1 diabetes are twice as likely to have been fed soy formulas as those fed all other foods This confirms concerns based on animal studies raised in the 1980's and 1990s by Health Canada researcher Dr Fraser Scott and led to the American Academy of Pediatrics issuing their warning to pediatricians against any use of soy based formulas.
Researchers at Cornell University Medical College said that children who got soy formula were more likely to develop thyroid disease and that twice as many diabetic children had received soy formula in infancy as compared to non-diabetic children. In fact, in other countries such as Switzerland, England, Australia and New Zealand, public health officials recommend highly restricted medically monitored use of soy for babies and for pregnant women. Soy formula is a lifesaver for the 3 to 4 percent of babies allergic to cows milk, but it is so widely advertised that it is sold to 25% of the entire formula market.
Soybeans in fact contain a large number of dangerous substances.

One among them is phytic acid, also called phytates. This organic acid is present in the bran or hulls of all seeds and legumes, but none have the high level of phytates that soybeans do. These acids block the body's uptake of essential minerals like calcium, magnesium, iron and especially zinc. Adding to the high-phytate problem, soybeans are very resistant to phytate reducing techniques, such as long, slow cooking.
Soybeans also contain potent enzyme inhibitors. These inhibitors block uptake of trypsin and other enzymes that the body needs for protein digestion. Normal cooking does not deactivate these harmful "antinutrients," that can cause serious gastric distress, reduced protein digestion and can lead to chronic deficiencies in amino acid uptake.
Beyond these, soybeans also contain hemagglutinin, a clot promoting substance that causes red blood cells to clump together. These clustered blood cells are unable to properly absorb oxygen for distribution to the body's tissues, and cannot help in maintaining good cardiac health. Hemagglutinin and trypsin inhibitors are both "growth depressant" substances. Although the act of fermenting soybeans does deactivate both trypsin inhibitors and hemagglutinin, precipitation and cooking do not. Even though these enzyme inhibitors are reduced in levels within precipitated soy products like tofu, they are not altogether eliminated. Only after a long period of fermentation (as in the creation of miso or tempeh) are the phytate and "antinutrient" levels of soybeans reduced, making their nourishment available to the human digestive system. The high levels of harmful substances remaining in precipitated soy products leave their nutritional value questionable at best, and in the least, potentially harmful

Check this out

http://www.soyonlineservice.co.nz/

Mind you this is probably a one sided article from the beef industry.
 
I've read that soy can reduce testostrone in your body... One of the reasons it has been claimed to be a "good female protein"...... That never really made a lot of sense to me.

Good Post Ms. W.
 
cjas said:
I've read that soy can reduce testostrone in your body... One of the reasons it has been claimed to be a "good female protein"...... That never really made a lot of sense to me.

Good Post Ms. W.



:iggthumpu
 
I think the negative impact on the thyroid maybe soy's most harmful effect.
 
liftsiron said:
I think the negative impact on the thyroid maybe soy's most harmful effect.

That might explain why my husband's thyroid crashed. It never came back either :(
 
liftsiron said:
I think the negative impact on the thyroid maybe soy's most harmful effect.
Completely agree - especially when a huge percentage of people suffering from hypothyroid are women - but there they are drinking another soy latte :nono:
 
Ms.Wetback said:
Mind you this is probably a one sided article from the beef industry.
Exactly, oatmeal is nearly as high in phytates as Soy. Another fallacy is that soy protein produces estrogen. It doesn't. It contains phytoestrogen. This is a good thing, as phytoestrogens will bind to estrogen receptors as opposed to estrogens.

I wouldn't use soy as my only source of protein, but then again, I wouldn't use any one single protein as my only source of protein. If I was forced to choose one source of protein as my only source of protein, it would be egg protein. Soy protein has overall cardio-protective, leaning, and anti-cholesterol effects. Remember, Sergio and Arnold used Soy and casein proteins as whey wasn't available back then.
 
I swear, you cant eat anything anymore. An ortho doc I know is married to an oncologist and she wont let him eat ANYTHING. She is always on his ass about the stuff he eats because so many foods have been linked cancer. Good post.
 
Good post Ms.WB!

Soy foods contain trypsin inhibitors that inhibit protein digestion and affect pancreatic function. In test animals, diets high in trypsin inhibitors led to stunted growth and pancreatic disorders. Soy foods increase the body’s requirement for vitamin D, needed for strong bones and normal growth. Phytic acid in soy foods results in reduced bioavailabilty of iron and zinc which are required for the health and development of the brain and nervous system. Soy also lacks cholesterol, likewise essential for the development of the brain and nervous system. Megadoses of phytoestrogens in soy formula have been implicated in the current trend toward increasingly premature sexual development in girls and delayed or retarded sexual development in boys.

Soy isoflavones are phyto-endocrine disrupters. At dietary levels, they can prevent ovulation and may stimulate the growth of cancer cells. Eating as little as 30 grams (about 4 tablespoons) of soy per day can result in hypothyroidism with symptoms of lethargy, constipation, weight gain and fatigue.

Soy foods may stimulate the growth of estrogen-dependent tumors and cause thyroid problems. Low thyroid function is associated with difficulties in menopause.

Numerous animal studies show that soy foods cause infertility in animals. Soy consumption enhances hair growth in middle-aged men, indicating lowered testosterone levels. Japanese housewives feed tofu to their husbands frequently when they want to reduce his virility.

source: Soy on line
 
Last edited:
Exactly, oatmeal is nearly as high in phytates as Soy. Another fallacy is that soy protein produces estrogen. It doesn't. It contains phytoestrogen. This is a good thing, as phytoestrogens will bind to estrogen receptors as opposed to estrogens.

No it's not a good thing, it would be a good thing if phytoestrogen acted as estrogen receptor antagonists or even negative modulators, but instead they act as estrogen receptor agonists, they mimic and potentate the the effects of endo estrogen, which increases overall estrogen.

Interaction of Estrogenic Chemicals and Phytoestrogens with Estrogen Receptor ?
George G. J. M. Kuiper1, Josephine G. Lemmen, Bo Carlsson, J. Christopher Corton, Stephen H. Safe, Paul T. van der Saag, Bart van der Burg2 and Jan-?ke Gustafsson3

Center for Biotechnology and Department of Medical Nutrition (G.G.J.M.K., J.-?.G.), Karolinska Institute and KaroBio AB (B.C.) Huddinge, Sweden; Hubrecht Laboratory, Netherlands Institute for Developmental Biology (B.v.d.B., P.T.v.d.S., J.G.L.) Utrecht, The Netherlands; Chemical Industry Institute of Toxicology (J.C.C.), Research Triangle Park, North Carolina; Department of Veterinary Physiology and Pharmacology (S.H.S.), Texas A&M University, College Station, Texas 77843-4466

Address all correspondence and requests for reprints to: Dr. George Kuiper, Center for Biotechnology, NOVUM, S-14186 Huddinge, Sweden. E-mail: [email protected].

The rat, mouse and human estrogen receptor (ER) exists as two subtypes, ER{alpha} and ER?, which differ in the C-terminal ligand-binding domain and in the N-terminal transactivation domain. In this study, we investigated the estrogenic activity of environmental chemicals and phytoestrogens in competition binding assays with ER{alpha} or ER? protein, and in a transient gene expression assay using cells in which an acute estrogenic response is created by cotransfecting cultures with recombinant human ER{alpha} or ER? complementary DNA (cDNA) in the presence of an estrogen-dependent reporter plasmid.

Saturation ligand-binding analysis of human ER{alpha} and ER? protein revealed a single binding component for [3H]-17?-estradiol (E2) with high affinity [dissociation constant (Kd) = 0.05 - 0.1 nM]. All environmental estrogenic chemicals [polychlorinated hydroxybiphenyls, dichlorodiphenyltrichloroethane (DDT) and derivatives, alkylphenols, bisphenol A, methoxychlor and chlordecone] compete with E2 for binding to both ER subtypes with a similar preference and degree. In most instances the relative binding affinities (RBA) are at least 1000-fold lower than that of E2. Some phytoestrogens such as coumestrol, genistein, apigenin, naringenin, and kaempferol compete stronger with E2 for binding to ER? than to ER{alpha}. Estrogenic chemicals, as for instance nonylphenol, bisphenol A, o, p'-DDT and 2',4',6'-trichloro-4-biphenylol stimulate the transcriptional activity of ER{alpha} and ER? at concentrations of 100-1000 nM. Phytoestrogens, including genistein, coumestrol and zearalenone stimulate the transcriptional activity of both ER subtypes at concentrations of 1?10 nM. The ranking of the estrogenic potency of phytoestrogens for both ER subtypes in the transactivation assay is different; that is, E2 >> zearalenone = coumestrol > genistein > daidzein > apigenin = phloretin > biochanin A = kaempferol = naringenin > formononetin = ipriflavone = quercetin = chrysin for ER{alpha} and E2 >> genistein = coumestrol > zearalenone > daidzein > biochanin A = apigenin = kaempferol = naringenin > phloretin = quercetin = ipriflavone = formononetin = chrysin for ER?. Antiestrogenic activity of the phytoestrogens could not be detected, except for zearalenone which is a full agonist for ER{alpha} and a mixed agonist-antagonist for ER?. In summary, while the estrogenic potency of industrial-derived estrogenic chemicals is very limited, the estrogenic potency of phytoestrogens is significant, especially for ER?, and they may trigger many of the biological responses that are evoked by the physiological estrogens.




This article has been cited by other articles:


Home page
EndocrinologyHome page
B. R. Bhavnani, S.-P. Tam, and X. Lu
Structure Activity Relationships and Differential Interactions and Functional Activity of Various Equine Estrogens Mediated via Estrogen Receptors (ERs) ER{alpha} and ER{beta}
Endocrinology, October 1, 2008; 149(10): 4857 - 4870.
[Abstract] [Full Text] [PDF]
 

Trending

Back
Top